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ME/CFS patient serum induces metabolic changes in myoblasts, including excessive lactate, which indicates that PDH dysfunction underlies PEM and inadequate ATP generation [76]

This approach enabled efficient analysis of a large dataset collected during routine clinical care, allowing for a timely evaluation of L-carnitines potential benefits

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For instance, a review on the neurobiology of ALCAR, carried out by the National Center for Biotechnology Information (NCBI), stated that ALCAR slows the progression of mental decline and has an effect on mitochondria metabolism thus showing promise as a treatment for neurodegenerative diseases

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