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Overexpression of TRRAP, a constituent of several histone acetyltransferase complexes, increases mutp53 levels, whereas silencing TRRAP reduces mutp53 accumulation in lymphoma and colon cancer [43]

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Its interesting from a scientific standpoint, but boy does it complicate the public health messaging. Prior to the CANDIDE study, Springer and his UCSF research group had done many studies in rats, which have similar cardiovascular responses to tobacco smoke as humans do, and which can be studied under tightly controlled experimental conditions