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CNS and Neurological Disorders-Drug Targets, 17 (8), 590594

Reville PK, MD, MPH, Kantarjian H, MD, Borthakur G, MD, Pemmaraju N, MD, DiNardo CD, MD, MSCE, Sasaki K, MD, PhD, Daver N, MD, Issa GC, MD, Ohanian M, DO, Montalban-Bravo G, MD, Short NJ, MD, Jain N, MD, Bhalla KN, MD, Jabbour EJ, MD, Ferrajoli A, MD, Takahashi K, MD, PhD, Rausch CR, PharmD, Malla R, RN, BSN, Marek K, Brandt M, BS, Popat UR, MD, Andreeff M , MD, PhD, Garcia-Manero G, MD, Konopleva M, MD, PhD, Ravandi F, MBBS, Kadia TM, MD

10.1016/j.freeradbiomed.2005.05.021 Free Radic

10.1016/j.jhsa.2008.07.003 14 ChenW.SunY.GuX.CaiJ.LiuX.ZhangX.et al (2021)

Therefore benefits of clorgiline treatment observed in the above studies may be due to reduced -adrenergic activation rather than reduced MAO-mediated metabolites

PI3K/Akt/mTOR activation Hits the central anabolic signalling cascade - the same pathway behind insulin's metabolic effects and most growth-factor-driven hypertrophy