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Inhibition of substance P/NK-1R interactions using the non-peptide NK-1R antagonist RP-67580 reduces the inflammatory response and the reactive astrogliosis associated with Trypanosoma brucei brucei infection of the CNS (Kennedy et al., 1997), further supporting a role for substance P/NK-1R interactions in neuroinflammation associated with eukaryotic parasite CNS infections

[ii] Ibid

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