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The link is there but literature is unclear on whether TMAO is a bystander or a mediator (Janeiro et al

crassiceps infection prevented the up-regulation of -catenin and CXCR2 expression, which are both markers associated with colitis-associated colorectal tumorigenesis [78]

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LPS, particularly its lipid A portion, is actually toxic

Any of these make an auto-immune cause of B12 deficiency (PA, also called autoimmune gastritis 3 ) more likely

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