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In summary, we have applied, for the first time, an untargeted adductomics approach in a setting of DNA repair deficiency, uncovering novel endogenous DNA adducts that are likely to contribute substantially to mutagenesis

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The high immunoactivity of MYC-tagged -catenin (MYC) in c-Met/-catenin-driven liver tumors and HA-tagged myr-AKT1 (HA) in AKT/-catenin-driven liver tumors implied their origin from the transfected cells (Fig