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We previously demonstrated that CD38 in skin fibroblasts was upregulated by inflammatory cytokines TNF-, IL13 as well as TGF- 24

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Notably, the inhibition of CD38s extracellular cyclase activity accounts for only 2% of the molecules total enzymatic activity when targeted by therapeutic antibodies ( An alternative view suggests that the in vitro conditions used in these studies might not adequately represent in vivo dynamics

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