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Notably, the diffusion of FITC-dextran was not observed, confirming that the excreted glucose was not the result of diffusion, but occurred via the glucose transporter (Supplementary Fig

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This was due to the impaired glycosylation of kaempferol by these enzymes to form kaempferol 3-O-glycosides that is a concurrent biochemical pathway for 4-HBA synthesis

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Collectively, these observations support the view that Pb toxicity constitutes a systems-level phenomenon driven by tightly interconnected physiological and metabolic adjustments