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D.DewhurstS.et al

Answer and interpretation This is probably valproic acid induced hyperammonia encephalopathy (VHE) VHE has been reported secondary to acute VPA overdose and in chronic normal therapeutic dosing, presenting as an acute and profound decreased level of consciousness, with or without increased seizure activity this is usually in association with an increased serum ammonia level (Mock and Schwetschenau, 2008) Patients at higher risk for VHE include those with liver impairment, on multiple anticonvulsants, or have other causes of increased ammonia production

However, this is just a scientific hypothesis that has not been verified in clinical trials [47]

Son rle phare est celui dune navette : elle aide certains acides gras entrer dans les mitochondries, o ils peuvent tre utiliss pour produire de lnergie