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Liver fibrosis is the major cause of morbidity and mortality in patients with CLD [1]

Some splicing changes introduce premature stop codons and presumably induce nonsense-mediated decay of the mRNA (leading to reduced synthesis and function), while others modulate protein sequences in important domains (Supplementary Table 3)

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doi: 10.1016/j.burns.2012.09.006

Subcellular characteristics of phospholipase A2 activity in the rat kidney