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As the in vitro kinase reaction shown, a truncation of Sec24C consisting of only the first 400 residue (C) rather than the remaining portion (N) was phosphorylated by VRK2 (Fig

Additionally, ALA exhibited neuroprotective effects by effectively inhibiting apoptosis, morphological changes, and inflammatory responses in BV2 (Figure 3A,B) [27], as well as regulating the Wnt/-catenin pathway in PC12 cells activated by A 2535 [28]

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Abstract Neuropeptide Y (NPY) is a neurotransmitter involved in stress response and immune modulation and its dysregulation may contribute to the pathogenesis of vitiligo by influencing melanocyte function and autoimmune responses

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