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Briefly, in a well described mechanism, paracetamol is broken down by membrane-bound cytochrome p450 enzymes such as CYP2E1, CYP1A2 and CYP3A4 to its reactive intermediate, NAPQI which is known to covalently bind to mitochondria and cause direct hepatic toxicity by prompting increased generation of ROS and reactive nitrogen species (RNS) that ultimately drive apoptosis and necrotic cell death [1, 72, 73]

Graphical Abstract The guides -base systematic review process (Hutton et al

Metabolism 92, 7181

Trace elements and oxidative stress indicators in the liver and kidney of the blue shark (Prionace glauca)