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Recipient mortality was monitored for up to five months [2]

The proposed mechanism involves controlling glutamate release in the synaptic cleft, modulating NMDA receptor stimulation, affecting excitatory amino acid transporter (EAAT)-mediated reuptake, and maintaining the glutamate/glutamine cycle within astrocytes

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The test is designed to measure spontaneous alternation, which is the capacity of a mouse to naturally explore an arm of the maze instead of returning to an arm they have more recently visited, as an indicator of working memory over a short period of time

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