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Glutamatergic transmission is controlled and regulated by astrocytic glutamate-glutamine shuttle, thereby providing a means by which astrocytes may contribute to the pathophysiology of addictive disorders through glutamatergic pathways

NPs, intravitreal implants, hydrogels, combinatorial systems, and minimally invasive techniques are under preclinical and clinical investigations, which act as safer and more efficient alternatives to combat ophthalmic diseases [24, 117]

Other potential regulators and pathways of ferroptosis, such as miR-137 [38], peroxiredoxin-6 (PRDX6) [39], activating transcription factor 4 (ATF4) [40], heat shock protein beta 1(HSPB1) [41], the mevalonate pathway, and the transsulfuration pathway [42] are emergent players in ferroptosis regulation (Fig

Serological studies are negative for ANA, anti-GBM, ANCA, MPO, PR3 and ASO

The accumulation of cellular debris and apoptotic cells may act as a source of autoantigens and cause inflammation

The structural and functional units of heteromeric amino acid transporters