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Patients suffering from mucormycosis in allogeneic HSCT recipients were found to have a severe iron overload compared with a matched control population (122)

NAD+-boosting agent nicotinamide mononucleotide potently improves mitochondria stress response in Alzheimers disease via ATF4-dependent mitochondrial UPR

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Iron overload also significantly increased the protein and gene expression of FTH/L and SLC40A1 in the iron metabolic pathway, which was rescued by theaflavins, indicating theaflavins' ability to inhibit ferroptosis by restoring iron metabolism (Fig

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Birele Z, Vimba PM, evenko A et al (2025) The Association of Plasma Selenium and Selenoprotein P Levels with Depression Severity and Anxiety Symptoms Among Medical Students in Latvia