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The restoration of mTOR signaling with Fer1 rescue treatment in the context of RSL3 effects, and the genetic knockdown of GPX4, reaffirm our in vitro findings that RSL3-mediated induction of ferroptosis, mTOR pathway suppression, DNA damage and failure of DNA repair, and autophagy are GPX4-dependent processes in thyroid cancer cells

10.1016/j.jcf.2023.07.001 296 ZamanK.CarraroS.DohertyJ.HendersonE

: GHRP-2 actions on somatotrophs

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Sun AY, Wang Q, et al