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Interestingly, we observed that both EGFR pathway- and PI3K-transformed cells, are addicted to RNF4 and VHL

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Therefore, cystine deficiency or the inhibition of xCT function leads to impaired GSH synthesis and promotes ferroptosis[3, 4, 85-88]

evidence suggests TMAO contributes to OA pathogenesis by promoting chondrocyte apoptosis and ECM degradation (Wang Y

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J Occup Med 36(8):849854 Soldin OP, Mattison DR (2009) Sex differences in pharmacokinetics and pharmacodynamics