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As the field continues to evolve, identifying the most effective combinations based on tumor genomic characteristics and immune status will be critical for maximizing the therapeutic potential of KRAS-targeted therapies [4][11]

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If satisfactory clinical response has not been achieved, plasma levels should be measured to determine whether or not they are in the usually accepted therapeutic range (50-100 mcg/mL)

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The main drawback of CPT1A blockage is the undesirable impact on non-tumor cells given the extensive tissue distribution of CPT1A

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