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On one hand, thalassemia-related oxidative stress may alter homocysteine metabolism: one report suggests that increased oxidative stress can stimulate cystathionine -synthase (CBS) activity, shunting homocysteine to cysteine and thus lowering plasma homocysteine levels in -thalassemia [4]

Within the TME, mitochondrial transfer has emerged as a sophisticated form of cell-to-cell communication, enabling cancer and non-malignant cells to exchange not just molecules, but entire organelles with bioenergetic, genetic, and signaling capacities [332, 445]