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It has been reported that the glucagon-like peptide 1 (GLP-1) analog exendin-4 lowers ERS and enhances protein folding to ameliorate Hcy-induced endothelial dysfunction in vivo and in vitro (132)

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However, high concentrations of ROS can inhibit glyceraldehyde-3-phosphate dehydrogenase, impeding glycolysis 32 , and this glucose metabolism is crucial for T cell activation and proliferation 33

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This effect was corroborated by decreased interaction between endogenous E2 and E3 components in HGC27 and MKN74 GC cells upon TGF1 treatment (Fig