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[DOI] [PubMed] [Google Scholar] Sonnewald, U

Its mechanism relies on spatiotemporal coordination among epidermal cells, immune cells, and fibroblasts through extracellular matrix (ECM) remodeling and signaling factor cascades, involving multi-dimensional molecular interaction networks such as gene expression regulation, metabolic reprogramming, and microenvironmental homeostasis

The transepithelial resistance was recorded every 10 s using a CVC6 clamp device (Fiebig, Berlin Germany) 49 by measuring voltage deflections induced by injection of 20 A symmetrical square pulses for 0.4 s

Mutant p53 drives cancer chemotherapy resistance due to loss of function on activating transcription of PUMA

Sies et al

Hematocrit shifts are not a documented LR3 IGF-1 effect, but a baseline panel is the cheapest way to catch unexpected changes