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3.4 SIRT1s role in altered intercellular communication and inflammation Aging impairs intercellular communication within the liver, especially among hepatocytes, hepatic stellate cells (HSCs), Kupffer cells, and liver sinusoidal endothelial cells (LSECs) (Wan et al., 2022)

Although familial clustering has been reported, cryptorchidism is likely polygenic, with no single causative gene identified [54, 55]

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Enzymes such as FSP1 and MBOAT1/2 have been identified as important contributors to these pathways, suggesting a broader and more complex regulatory network of ferroptosis beyond the conventional System Xc -GSH-GPX4 axis (Figure 1 offers a comprehensive depiction of the critical molecular mechanisms underpinning ferroptosis