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Of course similar effects of disulfide "clipping" on secondary structure can be expected in case of every type of secondary structure

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Mechanistic model and therapeutic implications for SLE The pathogenesis of SLE is closely linked to autoantigen exposure resulting from defective DNA repair (150)

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Cell Proliferation/Viability Assay WB WB WB WB All MEK Isoforms More Biological Activity 50 & Target [1] In Vitro In BRAF mutant SK-MEL-28 cells and KRAS mutant HCT116 cells, Trametinib (GSK1120212;JTP-74057) DMSO solvate causes dose-dependent inhibition of ERK1/2 phosphorylation as well as dose-dependent growth inhibition

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