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Ovarian injury caused by CP leads to the release of damage-associated molecular patterns (DAMPs), which activate pattern recognition receptors (PRRs) such as Toll-like receptors, NOD-like receptors, and others on immune and non-immune cells (Ma et al

The enhancement of migratory capabilities, driven by epithelialmesenchymal transition (EMT) processes, is facilitated through ROS-mediated pathways involving MAPK/NF-B, as induced by the mitochondrial Lon protease (Kuo et al

Proper functioning of GABA is associated with the action of two main receptors, ionotropic GABA-A and metabotropic GABA-B

2024), which is especially the case for GSH metabolism

CHAC1 maintenance of matrix cysteine under limiting conditions could also support the generation of H 2 S and subsequent production of hydropersulfides, which potently mitigate lipid peroxidation 81

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