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In addition, studies on the role of PCSK9 in different cells involved in at all stages of the plaque have supported that the effect of PCSK9 on atherosclerotic development is beyond the LDLR degradation, and occurs by crosstalking with other molecular mechanisms ranging from the activation of inflammatory pathways to apoptosis of ECs and migration of VSMCs

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discovered that the activation of Nrf2 by ADAMTS-13 could inhibit ferroptosis to ameliorate cisplatin-induced AKI (Meng et al., 2021)

292 Deficiency of skeletal muscle cystathionine -lyase leads to insulin resistance, ferroptosis, and obesity, resulting in hyperglycemia and skeletal muscle injury in high-fat diet mice

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