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Description

Notably, we observed increased fibrosis in Aldh1l2 KO mice at day 4 following caerulein treatment and a delay in the regeneration of acinar cells at day 7, consistent with a role for re-expression of ALDH1L2 in the recovery from pancreatic damage (Extended Data Fig

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Studies involving humans show that mTBI, which may involve axonal damage or dysfunction as detected by diffusion tensor imaging, is associated with measurable decreases in CMRglc (Gross et al., 1996

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British Journal of Haematology 121, (5) (Jun): 749-57

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The interpretation that increased sPAF-AH activity caused an increased risk of atherosclerotic cardiovascular disease (ASCVD) led to the development of selective sPAF-AH inhibitors and their clinical trials (425)

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