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Willeit J

doi: 10.15232/pas.2017-01695 51 PateRTCardosoFC

We therefore concluded that the disulfide bond formation in the metallated SOD1 was facilitated, in the absence of CCS1, by ROS generated via the active-site Cu 2+ -catalyzed CSH oxidation

The pathological function of -Klotho in POF remains inadequately elucidated

It was effectively applied either immediately (prophylactically) after paracetamol challenge or (therapeutically) administered after a 3 h period had elapsed (note, the maximum depletion of hepatic glutathione occurred 3 h after acute paracetamol dosing (37))