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Specifically, improved treatment requires research scientists, clinicians and biomedical engineers to (i) Determine the quantitative relationships between risk factors, genetic susceptibility and molecular mechanisms that underlie cellular interactions in the front and the back of the eye, (ii) Develop standardized tests to define where the optic neuropathy in a particular patient falls in the spectrum between normal aging and glaucoma, (iii) Develop novel technologies (such as optical coherence tomography) for early diagnosis (iv) Improve the efficacy of pharmacological agents that target TM while eliminating side effects and (v) Protect the pressure sensitive targets within the trabecular meshwork, retinal ganglion cells and glia

Furthermore, the retardation of the suicidal radical coupling between the W251 residue and the monolignolic radical was attempted by supplementing the acidic microenvironment around the W251 residue to engineer radical-robust LiPH8

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5.1 Targeting lipid metabolism in treatment Inhibiting the de novo lipogenesis (DNL) pathway has become an important strategy for cancer treatment (192).FASN, the rate-limiting enzyme in DNL, is closely associated with tumor cell proliferation, metastasis, and chemotherapy resistance, making it a key target for inhibiting lipid synthesis (56)

[DOI] [PMC free article] [PubMed] [Google Scholar] 33.Suthar SK, Bansal S, Narkhede N, et al