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Firstly, mitochondrial oxidative phosphorylation (OXPHOS) provides the ATP necessary for collagen biosynthesis, post-translational modifications, proteoglycan synthesis, ECM assembly, and fibroblast migration during tissue repair [2,5,12]

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Among these, TGF- signaling is widely recognized as a central mediator of fibrotic progression and has emerged as a key therapeutic target [33, 36]