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Beyond its contribution to the global cancer burden, EBV has been linked to autoimmune conditions such as rheumatoid arthritis and multiple sclerosis 6,7,8,9,10

We show that replacing the R140, N141, L142 and W145 residues with alanine or deleting residues 129148 of the disordered linker region of STX7 decreased binding to Munc13-4 in TR-FRET assays (Fig

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