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The central role of glutathione in the pathophysiology of human diseases

Furthermore, we confirm that the initial glutathionylation of the glutaredoxin moiety and not the glutathione transfer to the roGFP2 moiety is rate-limiting for the oxidation of fusion constructs at physiological or low micromolar GSSG concentrations, thus allowing glutaredoxin structure-function analysis in vivo

The effect of tripeptide-copper complex on healing of ischemic open wounds

Kits available are: HOOK GST Protein Purification (Bacteria): Isolation from bacteria

The PCA was weighted by group size

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