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Figure 5 4 Promising therapeutical reagents for SLE by inhibiting ferroptosis Given that several pathophysiological features of ferroptosis, including iron accumulation, mitochondrial dysfunction, overproduction of lipid peroxidation and suppression xCT-GPX4 pathway are potentially implicated in SLE progression, reversing those features may be beneficial to SLE disease

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711 Cancer cells depend on these metabolic hubs to fulfill their heightened energy requirements and maintain ROS levels, which are crucial for their proliferation, migration, invasion, and metastasis