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doi: 10.1111/j.1365-2443.2010.01473.x, 228

doi: 10.1016/J.IJBIOMAC.2019.12.159 315 ZhongZ.NiimiM.TobisaM.IdotaS.IshiiY

The application proposes a novel mechanism of action of the disclosed combination that stabilizes infected cells during viral infection resulting in higher cell survival in in- vitro demonstrations of the anti-viral effect of the present invention [Para 1 8] Oxidation stress occurs when the balance between the production and the disposal of reactive oxygen species (ROS) favors the production of excess ROS, also known as free radicals

Current strategies can be broadly categorized into three approaches: (1) direct inhibition of HMGB1 to block its release or signaling via TLR4/RAGE

It has been demonstrated that the administration of DATS effectively inhibited the rise in ROS level caused by the expression of TDP-43 and enhanced cell viability by promoting the Nrf2 pathway

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