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This synthetic variant exhibits reduced binding to IGF binding proteins, allowing enhanced bioavailability and systemic activity [2]

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created an innovative dual-target c-MET/PD-1 antibody (BsAb) that blocks HGF-driven cell growth, movement, and apoptosis prevention, and prevents HGF-induced autophosphorylation at c-MET Tyr1234/1235 locations, protein kinase B (AKT), and extracellular signal-regulated kinases (ERK1/2)