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In cardiac pathophysiology models, Pue attenuates myocardial hypertrophy via PPAR/PGC-1-mediated upregulation of CPT1a/b, enhancing fatty acid oxidation (85)

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The association of wild-type BRD4 ET with DNA was largely unaffected in the presence of ASXL1 EBM in EMSA, confirming that the two binding sites located on the opposite sides of BRD4 ET do not overlap (Fig

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