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(1994) The ratio between serum levels of insulin-like growth factor (IGF)-I and the IGF binding proteins (IGFBP-1, 2 and 3) decreases with age in healthy adults and is increased in acromegalic patients

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demonstrated that ZDHHC5-mediated palmitoylation of beclin 1 serves as a core mechanism for maintaining neuronal autophagy, and its deficiency in AD murine models impairs autophagic function, exacerbating proteotoxic deposition, thereby leading to neurodegeneration, long-term potentiation (LTP) deficits, and memory deficits (167)