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A deficiency can result in severe neurological problems, including memory loss, depression, and significant nerve damage

Transcription factors regulate SLC7A11 expression and thus modulate ferroptosis

Human proximal tubule epithelial cells modulate autologous dendritic cell function

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As a further example, corticotropin-releasing factor is secreted from the hypothalamus under stress and, together with neurotensin, can stimulate brain mast cells to release inflammatory and neurotoxic mediators that disrupt the BBB, stimulate microglia, and cause focal inflammation [527, 528]