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The aetiological agents of CD are gluten immunogenic peptides (GIPs) derived from Triticeae prolamins (Rodrguez-Ramrez et al, 2024), which, unlike most food proteins, are not fully digested by the peptidases from the gastrointestinal tract (GIT) due to their high proline content (roughly 35%) (Bonner et al, 2025)

4.1 Oxidative stress and mitochondrial damage Oxidative stress and subsequent mitochondrial damage are the key pathological initiating factors of toxicity-induced hepatic metabolic barrier damage

doi: 10.1016/j.matpr.2021.03.384 116 PiatekM.OBeirneC.BeatoZ.TackeM.KavanaghK

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MRI with T1 and T2 mapping can detect copper deposition with greater sensitivity than ultrasound, though cost limits its use as a screening tool [7]

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