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Under basal conditions, NIK is constitutively degraded via a complex involving TNF receptor-associated factor 2 (TRAF2), TNF receptor-associated factor 3 (TRAF3), and cellular inhibitors of apoptosis proteins (cIAP1/2) [82]

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Extracellular matrix remodeling through endocytosis and resurfacing of Tenascin-R

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J Biol Chem 254(16):75587560 Hallberg E, Rydstrom J (1989) Selective oxidation of mitochondrial glutathione in cultured rat adrenal cells and its relation to polycyclic aromatic hydrocarbon-induced cytotoxicity

Accordingly, in dysferlin-deficient muscles, membrane resealing is substantially compromised, leading to the accumulation of damaged muscle fibers and degeneration of muscle tissue, eventually resulting in the development of progressive muscular dystrophy (Bansal et al, 2003)