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ROS and Rheumatoid Arthritis Due to the physiological function of ROS, high levels of reducing equivalents and excessive ROS scavenging may lead to damage of the opposite type to oxidative stress, sometimes referred to as reductive (113) or antioxidative (114) stress

Evidence from preclinical studies highlights stronger tendon-to-bone healing and improved structural integrity compared to untreated conditions, concluding with the reference to BPC-157

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Entgiftungsphase: Die Schadstoffe werden aus den Zellen geholt und ausgeschieden