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Within the intricate framework of psoriasis pathophysiology, a noteworthy metabolic alteration is observed in T cells

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Similarly, hearts from transgenic mice overexpressing GPx1 had preserved mitochondrial and cardiac function in the presence of doxorubicin, with increased efficiency of mitochondrial respiration and better contractility and diastolic function compared wild type hearts exposed to doxorubicin [44]

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Although research into this syndrome is increasing, the aetiology is not clear, although the symptoms are thought to be due to nociceptive or neurogenic mechanisms