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571 Forsythoside A inhibits ferroptosis through Nrf2/GPX4 axis activation, thereby alleviating Alzheimers-like pathology

In this review, we describe the most recent advances concerning the role of GPX4 and, more generally ferroptosis in the myeloid lineage and in the emergence of AML

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Furthermore, transgenic mice overexpressing GTPCH treated with streptozotocin (STZ) are able to maintain endothelial function [19]