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IL-22 pretreatment significantly upregulates hepatic LC3-II(the PE-conjugated form of light chain 3) and phosphorylation of AMP-activated kinase (p-AMPK) in APAP-treated mice, and IL-22-mediated LC3-II conversion and protection against APAP-induced cytotoxicity are impaired when p-AMPK is blocked by compound C (an AMPK inhibitor) [203]

Redox Signal 29 (16), 15891611

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Several studies have established the receptor for complement molecule C3a (C3aR) as a promising therapeutic target after cerebral ischemia

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