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In summary, our findings suggest that lipolysis in M2 macrophages alters the cellular lipid profile, with FA 24:1 serving as a crucial metabolic substrate that enhances mitochondrial respiration and supports efferocytosis

Castoldi, A

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Animal-model studies report improved tensile strength of healed wounds, reduced scar volume, and accelerated closure with GHK-Cu treatment

Inositol, a pseudo-vitamin, may influence cellular signaling for lipid breakdown