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It also attacks the cell that causes autoimmune conditions by reducing free radicals

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In asthma, IL-13 promotes goblet cell hyperplasia and MUC5AC expression via the IL-4 receptor (IL-4R)/signal transducer and activator of transcription 6/SAM-pointed domain-containing E26 transformation-specific transcription factor pathway while suppressing forkhead box A2, a negative regulator of MUC5AC transcription [17]

In preclinical studies, several combination therapy strategies have achieved breakthrough progress and demonstrated significant potential for translation into clinical applications

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