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The formation of methemoglobin from hemoglobin within red cells is an ongoing oxidative process that results from exposure of hemoglobin to a variety of highly reactive molecules (oxygen free radicals), produced during normal cell metabolism [4]

This decrease in GCL expression directly affects glutathione synthesis, probably related to the observed increase in transforming growth factor (TGF-) levels in patients with Type 2 diabetes ( In general, glutathione plays a crucial role as an antioxidant in protecting the body from oxidative stress

In addition, compounds targeting mitochondrial nitric oxide radicals can effectively inhibit ferroptosis in various cells, indicating that mitochondrial lipid peroxidation plays a key role in ferroptotic OS (Krainz et al., 2016)

In some cases, researchers can trace the problem to oxidative stress

SGLT2 inhibitors act independently of SGLT2 to confer benefit for HFrEF in mice

The overlapping pathway analysis for the BALF comparison of sarcoidosis and controls identified complex networks with a large number of nodes (Fig