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Disclosure of interest: nothing to discloser E-Posters - ACUTE ISCHEMIC STROKE MANAGEMENT 01.00 - ACUTE ISCHEMIC STROKE MANAGEMENT - 01.01 - ACUTE MANAGEMENT - Neither thrombolysis nor thrombectomy P2109 - ESOC25-1903 TRIIODOTHYRONINE TREATMENT REDUCES STROKE VOLUME AND IMPROVES NEUROLOGICAL FUNCTION IN A MOUSE MODEL OF ISCHEMIC STROKE Daniel Ullrich 1,2 , Heike Heuer 2,3 , Steffen Mayerl 2,3 , Dagmar Fhrer 2,3 , Christoph Kleinschnitz 1,2 , Friederike Langhauser 1,2 1 Department of Neurology, University Hospital Essen, Germany, Essen, Germany, 2 Center for Translational Neuro- and Behavioural Sciences (C-TNBS), University Hospital Essen, Germany, E, Germany, 3 Department of Endocrinology, Diabetes and Metabolism, University Hospital Essen, University Duisburg-Essen, Germany, Essen, Germany Background and Aims: Thyroid hormones affect the central nervous system's response to various stimuli such as ischemic stroke

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In conclusion, as shown in Fig S1, the activation of eNOS by BPC 157 is mediated through multiple regulation processes including the promotion of VEGFR2 activation and endocytosis, the activation of positive regulator AKT, the reduction of the interaction of negative regulator Cav-1 with eNOS

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