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There may be a number of sources of this oxidative stress, including mitochondrial superoxide production, ischemia-reperfusion mechanisms and auto-oxidation of catecholamines

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doi: 10.3389/fpls.2018.01944 Received 28 September 2018 Accepted 13 December 2018 Published 09 January 2019 Volume 9 - 2018 Edited by Francisco Javier Cejudo, Universidad de Sevilla, Spain Reviewed by Peter J