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Our results suggest that the pharmacological inactivation of HIF-1, CREB, p53 or NRF2 could be beneficial for the treatment of obesity and type 2 diabetes in humans

NchelJGhatakSZukAVIllerhausAMrgelinMSchnbornKet alTGFB1 is secreted through an unconventional pathway dependent on the autophagic machinery and cytoskeletal regulators

Hyperandrogenism is a fundamental component of the pathophysiology of PCOS and primarily drives various phenotypic features, including follicular dysplasia, impaired ovulation, endometrial disorders, and metabolic dysfunction 20,21,22

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RNA-seq analysis provided insights into the molecular mechanisms by which EW exerts its neuroprotective effects