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10.1038/s41422-021-00582-x 145 ParkH.KamT.DawsonV

Taken together, these data show that although ABCC1 did not contribute to the acquired resistance to Venetoclax, inhibition of ABCC1 can restore Venetoclax sensitivity in resistant AML cells

Postulated mechanisms of metformin action, including AMPK activation and complex I inhibition, have been demonstrated in the setting of a consistent pleiotropic activity of this drug, suggesting that several molecular targets could be involved (Patil et al., 2014

Animal model: Protection and repair of intestinal epithelial physical barrier by IGF-1 LR3 Animal models are closer to the physiological environment in vivo and can comprehensively evaluate the impact of IGF-1 LR3 on the physical barrier of intestinal epithelium